Alcohol use disorder (AUD) is a chronic disease characterized in part by excessive intake
of ethanol (EtOH) and repeated relapsing events. Research on the neurobiological underpinnings that regulate (EtOH) intake and EtOH-seeking behaviors is essential for the development of treatments in the future. Previous literature has shown that the centrally-projecting Edinger Westphal nucleus (EWcp) is sensitive to EtOH, and may also regulate EtOH intake. The aim of this dissertation was to examine the role of the centrally-projecting Edinger-Westphal (EWcp) nucleus in EtOH-seeking behaviors.