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Abstract
Alcohol use disorders (AUDs) are a leading cause of preventable death and illness that contribute significant social and economic costs to society. Genetic factors contribute substantially to AUD development but identifying and characterizing their phenotypic expression at the cellular level has been incomplete. One of the greatest challenges in treating problem alcohol (ethanol; EtOH) use is our poor understanding of the diversity of factors that promote sensitivity to EtOH reward and intoxication.