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Abstract

Fetal Alcohol Spectrum Disorder (FASD) encompasses conditions caused by prenatal alcohol exposure, leading to lifelong cognitive and behavioral impairments. Current diagnosis relies on physical and neurological indicators, often identified years after birth, delaying intervention. Early detection is critical to reduce adverse outcomes, yet remains challenging due to symptom variability and overlap with other disorders. This research explores novel approaches for early FASD diagnosis by examining cerebral cortex development, a period highly sensitive to alcohol exposure and essential for future behavioral function. Findings aim to improve early identification strategies and inform therapeutic interventions during critical developmental windows.

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